OK,
I'll take it;
The damage from exploratory surgery with a needle in vain attempt to solve the decompression problem is the quick-temporary fix on the day of collection to get the fish sold.
At this point, perhaps no needle is worse then using one.
Buyers are usually city people who look for aesthetics to choose fishes to buy. They look for pefect fins and buy pretty fishes with nice fins.
The hidden, slow growing secondary infections that often arise later begin on the inside where the damage is not so apparent.
Days later the fish get exported and arrive clean as a whistle. This is because they have not eaten a thing for a week at this point.
Once in America, they progress thru the system and the race is on to sell it before it becomes visably thin and unsaleable..
The fishes survival instincts may push it to sample food and perhaps ingest it...but sadly, at the same time, the budding infection from the inside is working against it.
The infection may even be amplified by food interference. The fish may then stop attempts to feed.
Then before long you hear the age old hobbyists lament...
"He looked good and then just died for no reason.!"
The lucky ones may be the ones that were collected shallower water,benefited from at least some decompression, were smaller fishes [ small fishes decompress faster] and perhaps suffered less holes in the gut from needles.
So like borbonius and ventralis anthias and most genicanthus, enough survive to prime the pump and keep people trying.
Every species has stories of its running the guantlet to market. This tale of the big kingii is but one but stands out because he is a rare fish.
The big kingii was brought up w/out enough decom and damage was already done the day it happened.
He was damaged goods from the initial sale.
Short of taking him back down to 30 feet and restarting the decompression process, you would be left w/ the surgical venting by a practiced needle as the only option.
Steve